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Smokers' genes may helppredict whether they'll respond to drug treatments for nicotine addiction, anew study indicates. Worse cases may lead patients to take generic Advair available in various pharmacies. "People with thehigh-risk genetic markers smoked an average of two years longer than thosewithout these high-risk genes, and they were less likely to quit smokingwithout medication," study first author Dr. Li-Shiun Chen, an assistantprofessor of psychiatry at Washington University of Medicine in St. Louis, saidin a university news release. "The same gene variantscan predict a person's response to smoking-cessation medication, and those withthe high-risk genes are more likely to respond to the medication," Chensaid. In the clinical treatmentstudy, smokers with the high-risk variants were three times more likely thanthose without the variants to respond to treatments such as nicotine gum,nicotine patches, the antidepressant bupropion (Wellbutrin is one brand) andother drugs used to help people stop smoking. "Smokers whose geneticmakeup puts them at the greatest risk for heavy smoking, nicotine addiction andproblems kicking the habit also appear to be the same people who respond most robustlyto pharmacologic therapy for smoking cessation," senior investigator Dr.Laura Jean Bierut, a professor of psychiatry, said in the news release. The gene variants in thisstudy aren't the only ones involved in whether a person smokes, becomes addictedor has difficulty quitting, but they are an important part of the overallnicotine-addiction puzzle, the researchers said. "These variants make avery modest contribution to the development of nicotine addiction, but theyhave a much greater effect on the response to treatment," Bierut said."That's a huge finding." Thus, to buy Advair is cut to minimum. Smoking, particularly of cigarettes, is by far the main contributor tolung cancer. Cigarette smoke contains over 60 known carcinogens, including radioisotopesfrom the radon decay sequence, nitrosamine, and benzopyrene. Additionally,nicotine appears to depress the immune response to malignant growths in exposedtissue. Across the developed world, 91% of lung cancer deaths in men during theyear 2000 were attributed to smoking (71% for women). In the United States,smoking is estimated to account for 87% of lung cancer cases (90% in men and85% in women). Among male smokers, the lifetime risk of developing lung canceris 17.2%; among female smokers, the risk is 11.6%. This risk is significantlylower in nonsmokers: 1.3% in men and 1.4% in women. Women who smoke (former smokers and current smokers) and take hormonetherapy are at a much higher risk of dying of lung cancer. In a study byChlebowski et al. published in 2009, the women taking hormones were about 60%more likely to die of lung cancer than the women taking a placebo. Notsurprisingly, the risk was highest for current smokers, followed by pastsmokers, and lowest for those who have never smoked. Among the women who smoked(former or current smokers), 3.4% of those taking hormone therapy died of lungcancer compared to 2.3% for women taking the placebo.
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